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Mouse profilaggrin helps in skin cell differentiation and may be involved in calcium signaling.

The conclusion introduces a new way to classify skin cysts using their shape and genetic markers.

Two fibroblast types shape skin structure and repair differently.

Stem cells in eccrine glands could be used for regenerative medicine.

Scientists found a new, less invasive way to get stem cells from horse hair for veterinary medicine.

Basal stem cells in the skin have distinct types that are crucial for skin structure and health.

Pmg-1 and Pmg-2 are new genes important for skin and mammary gland development.

Sox2-expressing cells can help grow hair and heal skin.

Hoxc genes control hair growth through Wnt signaling.

Identical twins had different symptoms because one had more cells with an extra chromosome fragment in different tissues.

TheDES improve drug delivery through the skin but need more safety checks.

ES cell-derived stem cells can help regenerate skin and form gland-like structures.

A wide range of proteins are integrated into the skin's protective layer.

Ethosomes are a promising way to deliver drugs through the skin.

Human dermal papilla cell vesicles can reduce skin fibrosis in mice.

Scientists found markers called CD34 and CD200 that help identify stem cells in mouse and human hair follicles.

Combining DHT and EDC improves the strength and stability of PADM scaffolds for tissue engineering.

Tight junctions create a barrier in pig hair follicles that controls what can enter the skin.

Hair follicle cells resist turning into skin cells.

Deleting a specific protein in skin cells disrupts normal hair growth and development.

Secreted inhibitors of Wnt and IGF signaling control hair and tooth development, creating species-specific patterns.

Different markers are found in stem cells of the scalp's hair follicle bulge and the surrounding skin.

A genetic defect causes males in some Mediterranean populations to be born with ambiguous genitalia and develop male traits at puberty.

Epidermal β-catenin activation changes the dermis by signaling different fibroblast types.

Hairless mice have longer hair follicles and abnormal structures during the catagen phase.

Different harmful mutations in the CDH3 gene cause HJMD, but symptoms vary among individuals.