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BTNL2 helps protect hair follicles from immune attacks, which could aid in treating alopecia areata.

The 2012 criteria are better for diagnosing atypical lupus cases.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Certain genetic markers can indicate higher or lower risk for systemic lupus erythematosus.

Sweet syndrome can be the only sign of hairy cell leukemia relapse.

Type-2 immunity may influence skin diseases and could be targeted for treatment.

The document concludes that DAB389-IL2 is promising for treating refractory cutaneous T-cell lymphoma, but more research is needed on its effectiveness and side effect management.

Innate lymphoid cells type 1 may contribute to alopecia areata.

LHX2, with other markers, can identify hair placodes in rats.

Alopecia areata may be caused by antibodies targeting specific hair follicle proteins, hindering hair growth.

The SNP rs6457452 is linked to a higher risk of alopecia areata in Koreans.

A new mutation in the KRT86 gene was found to cause the hair disorder monilethrix in a Han family.

A specific gene variant may increase the risk of developing Alopecia Areata.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

The Fas/FasL pathway may play a role in alopecia areata.

Targeting specific T cells may help treat alopecia areata.

A new gene mutation is linked to monilethrix in the studied family.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

The role of γδT-cells in causing alopecia areata remains unclear.

BST2 is highly expressed in certain immune cells in alopecia areata, suggesting a role in the disease.

A specific type of immune cell plays a key role in causing alopecia areata and could be a target for treatment.

The rs231775 genetic variant is linked to a higher risk and severity of Alopecia Areata in males.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

Langerhans cells are crucial for skin immunity and allergic reactions.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

A new mutation in the hHb1 keratin gene is linked to the hair disorder monilethrix.

Alopecia areata patients have higher levels of certain immune receptors, suggesting new treatment possibilities.

mHa2 and mHa3 keratins have different structures and roles in mouse hair and tongue tissues.

A specific gene variant (rs4833095) is linked to a higher risk of alopecia areata in Koreans.