Search

everythinglearnresearchcommunity

for

Search:↓

Four genetic risk areas related to male-pattern baldness were identified, with WNT signaling playing a role in its development.

Different STUB1 gene mutations cause varied symptoms in autosomal recessive ataxias.

A mutation in the MAP2 gene causes reduced hair follicle density, leading to hairlessness.

Black hairy tongue can be an early sign of graft-versus-host disease after stem cell transplants.

A patient with Ivemark syndrome developed mixed type vitiligo after a hepatitis C infection, showing different treatment responses and immune cell involvement in the skin.

Researchers made a detailed map of gene activity for different parts of human hair follicles to help create targeted hair disorder treatments.

A rare combination of halo nevi, nonsegmental vitiligo, and early gray hair can occur together.

Variation in the TCHH gene affects wool curliness in sheep.

The new method found new shared genetic areas linked to both Type 2 Diabetes and Prostate Cancer.

KAP genes are crucial for hair development and show both shared and unique traits in humans, chimpanzees, and baboons.

Higher IL-37 levels are linked to more severe alopecia areata, but the gene variation doesn't affect disease risk.

Hair loss gene found on chromosome 3q26.

The EDAR gene greatly affects hair thickness in Asian populations.

Male-to-female skin grafts in mice are rejected due to sex-linked antigens.

Female donor to male recipient sex mismatch and positive ACA-IgG are key risk factors for vitiligo and alopecia areata in chronic GvHD patients.

Trichohyalin in hair can trigger immune attacks in alopecia areata.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Somatic BHD mutations are rare in Japanese renal tumors.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

New genotype linked to non-classical congenital adrenal hyperplasia found in Italian siblings.

The conclusion is that the variation in hair thinness in patients is mostly due to the amount of underdeveloped hairs, and treatments that thicken fine hairs might work for those with mild to severe conditions.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Different populations have distinct hair structures related to their ancestry.

The 2012 criteria are better for diagnosing atypical lupus cases.

Some drugs can cause rare but potentially deadly skin reactions, and early treatment and avoiding the drug again are key.

Chemotherapy and bone marrow transplant can cause permanent hair loss.