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Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

A woman with lupus and Kikuchi-Fujimoto disease improved with treatment.

A new CDH3 gene mutation was found in a Spanish patient with sparse hair and eye issues.

Higher levels of certain immune cells in hair follicles may contribute to alopecia areata.

ILC1 cells contribute to hair loss in alopecia areata.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

The conclusion is that the variation in hair thinness in patients is mostly due to the amount of underdeveloped hairs, and treatments that thicken fine hairs might work for those with mild to severe conditions.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

BTNL2 helps protect hair follicles from immune attacks, which could aid in treating alopecia areata.

High amphiregulin in the skin is a bad sign for acute graft-versus-host disease.

CK 5/6 expression in breast cancer is linked to negative hormone receptor status and higher tumor grade.

The document covers various dermatological treatments and conditions.

A 65-year-old man developed a serious skin cancer linked to a drug he was taking for a fungal infection after a bone marrow transplant.

TB and BCC tumors show similar follicular differentiation patterns.

Cancer patients treated with immune checkpoint inhibitors may develop alopecia, but some hair regrowth is possible with treatment.

Aggressive immunosuppressive treatment improved a woman's severe heart condition linked to autoimmune disease.

Lupus is a complex disease that requires personalized treatment because it varies greatly between individuals.

Expanded CD8+ T cells are linked to Alopecia Areata and may cause relapse after treatment.

miR-200c-3p could help diagnose and treat alopecia areata.

Mutations in the hairless gene cause hair loss and skin cysts in rhesus macaques.

cGVHD often severely affects the skin, causing rapid aging and other issues.

Targeting specific T cells may help treat alopecia areata.

A specific genetic trait in tumor cells is linked to longer survival without disease in certain lymphoma patients.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

A mutation in the IL2RA gene increases the risk of alopecia areata.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

New treatments for alopecia areata may target specific immune cells and pathways involved in hair loss.

Two new mutations in the CDH3 gene cause hair loss and vision problems in a young girl.