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A specific gene variant is linked to heart disease, increased heart muscle, curly hair, and thick skin on palms and soles.

Notch1 signaling is impaired in hidradenitis suppurativa, affecting skin and hair cells.

CD28 is a promising target for treating alopecia areata with belatacept.

CD28 is a promising target for treating alopecia areata with belatacept.

New mutations in the DSG4 gene cause a rare hair condition.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Malondialdehyde-modified DNA may trigger an immune response in alopecia areata patients.

Genetic testing can diagnose hair loss linked to DSG4 gene variants.

The boy's severe skin disorder is caused by two new mutations in his TGM1 gene.

Disease-specific tools better assess quality of life in alopecia areata patients.

A woman with chronic hepatitis C had a rare skin condition linked to her illness.

Certain KIR genes in Indian SLE patients are linked to disease severity and could be biomarkers.

Certain blood markers, especially MLR, can help diagnose alopecia areata.

Daclizumab may cause psoriasis-like skin problems in multiple sclerosis patients.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

LSD1 is essential for healthy skin development and creating the skin's protective barrier.

Targeting CD200 could be a new treatment for rheumatoid arthritis.

A mutation in the hHb3 gene is linked to the hair disorder monilethrix.

Skin disease activity scores can help decide when skin symptoms no longer affect the quality of life in patients with mild dermatomyositis.

miRNAs in the Dlk1-Gtl2 region may improve lamb fur quality.

Human placenta extract reduces inflammation and symptoms in atopic dermatitis.

The skin of both rat strains showed similar lectin binding patterns.

Scarring alopecia involves increased immune cells and specific gene changes near damaged hair follicles.

High amphiregulin in the skin is a bad sign for acute graft-versus-host disease.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

The Thr1022Ala variant in the hairless gene is not a disease-causing mutation.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

A genetic mutation in the hHa1 gene creates a smaller, but still functional, hair protein without causing hair problems.