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A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Dopamine affects coat color changes in agouti mice.

Sonic hedgehog signaling is needed for the development of touch-receptor cells in the skin, and the loss of Polycomb repressive complex 2 can lead to more of these cells.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Genetic testing for hairless gene mutations is crucial to correctly diagnose and treat atrichia with papular lesions.

The method allows for 3D tracking of hair follicle stem cells and shows they can regenerate hair for up to 180 days.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

The method accurately measures hormones and endocannabinoids in mice, showing gut microbiota diversity affects these levels and may influence stress and reproductive systems.

A gene mutation causes monilethrix in a Chinese family.

The rat model showed early perimenopause with regular cycles and low hormones, and late perimenopause with irregular cycles and higher hormone levels.

Diphencyprone initially increases mouse hair growth, then slows it, possibly due to changes in specific protein levels.

Mouse tail skin has different keratinization near hair follicles and scales.

Using a gene therapy with the Sonic Hedgehog gene helps mice regrow hair faster after losing it from chemotherapy.

Matriptase-2 helps control iron levels by suppressing hepcidin, and its deficiency can cause iron-deficiency anemia.

PP2Acα is essential for proper hair and skin development.

A mutation in the KRTHB5 gene causes hair and nail issues.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Removing GRK2 in skin cells causes hair loss similar to immune-related alopecia.

Genetically modified rats help reveal how vitamin D affects bone and skin health.

Gamma-ray exposure improves genome editing efficiency in mice using the i-GONAD method.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

H1 increases risk for neurodegenerative diseases, while H2 offers protection but is linked to other disorders.

Overexpressing the epigen gene in mice leads to enlarged sebaceous glands and greasy fur.

A mutation in the hHb3 gene is linked to the hair disorder monilethrix.

The study suggests that motor neurons created from stem cells of patients with spinal and bulbar muscular atrophy show signs of the disease, including changes in protein levels and cell functions.

The method quickly analyzes hair growth genes and shows that blocking Smo in skin cells stops hair growth.