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Hairless rats are good for testing anti-inflammatory drugs, similar to haired rats, without needing to remove fur.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

High levels of human keratin 16 in mice cause skin lesions and abnormal skin development.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

The study maps how genes are regulated during mouse hair growth.

New and known mutations in the hairless gene cause a hair loss condition called Atrichia with papular lesions.

Disrupting the FGF5 gene in rabbits leads to longer hair by extending the hair growth phase.

Overexpressing the epigen gene in mice leads to enlarged sebaceous glands and greasy fur.

Hair growth and pigmentation in mice involve specific stages crucial for research.

The gene for Marie Unna hereditary hypotrichosis is located on chromosome 8p21, near the hairless gene.

Mouse skin can produce and process serotonin, with variations depending on hair cycle, body location, and mouse strain.

AIRE-deficient rats developed severe autoimmune disease similar to APECED, useful for testing treatments.

A specific mutation in Kras causes abnormal tissue changes by making a cell signal continuously active, which disrupts normal cell coordination.

The fuzzy gene is crucial for controlling hair growth cycles.

MRI can effectively image skin structures noninvasively.

Overexpressing the glucocorticoid receptor in mice leads to abnormal skin development and reduced inflammation.

Two mouse mutants have defective hair cuticle cross-linking.

β-catenin, Shh, and Bmp signaling control hair follicle development.

Minoxidil boosts hair growth in genetically modified mice.

Animal and mathematical models help understand and develop treatments for alopecia areata.

Increasing miR-149 reduces hair follicle stem cell growth and hair development by affecting certain cell growth pathways.

Keratin 16 delays skin maturation and affects skin and hair development in mice.

Overexpression of Hoxc13 in hair cells causes hair loss and skin issues.

The HR protein's role as a repressor is essential for controlling hair growth.

Vitamin D receptor is crucial for starting hair growth after birth.

Ovalbumin–aluminum sensitization causes increased pain sensitivity and nerve changes in mice.