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Estrogen receptor α controls hair growth cycles and skin thickness in male mice.

Transplanted whisker follicles caused long hair growth on the spinal cords of mice.

Dexamethasone speeds up hair loss in mice, while cyclosporin A slows it down.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Minoxidil and PRP can help convert miniaturized hair follicles back to normal in male pattern baldness.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Minoxidil rejuvenates hair follicles and reduces aging signs in male pattern baldness.

B6J mice live longer before 24 months, but B6N mice live longer after; both strains show weight gain, increased food and water intake, and health issues as they age.

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

Whn is crucial for hair growth in certain areas by controlling a specific gene.

Scientists have found a way to create hair follicles from skin cells of newborn mice, which can grow and cycle naturally when injected into adult mouse skin.

The Lexington LaserComb helped regrow hair in mice with a condition similar to human hair loss.

New hair growth corticosterone levels are higher in diabetic mice, indicating long-term stress.

The scraggly mutation causes hair loss and skin defects in mice.

Minoxidil did not affect hair growth in newborn mice.

A new keratin gene was found in mice, explaining hair growth.

Old C57BL/6 mice with unsynchronized hair cycles show less melanin in their spleens.

Water-soluble minoxidil effectively promoted hair growth in mice.

Laser treatment helped regrow hair in mice by activating a key growth pathway.

Both gene and non-gene areas of DNA evolved to make some mammals hairless.

Researchers created a 3D-printed skin model that grew human hair when grafted onto mice by improving blood supply to the grafts.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

ODC transgenic mice can model human hair loss with skin lesions.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.