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A PTH-based treatment improved hair regrowth better than ruxolitinib in a mouse model of hair loss.

Both induced and spontaneous AA lymphocytes can cause alopecia areata in mice.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

Researchers found a new gene, hacl-1, that is active in mouse hair follicles during hair growth and may be important for hair biology.

CD25+ CD4+ Tregs and certain plasma proteins are linked to hair loss.

iNKT cells help develop and maintain healthy skin in young mice.

Patients with alopecia areata have higher levels of certain immune markers, suggesting new treatment targets.

IGF2BP3 and other m6A-related genes are linked to keloid formation and could be potential treatment targets.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

Ixekizumab may be an effective first treatment for a rare skin condition causing hair loss.

The study created special nanoparticles that effectively deliver an anti-inflammatory drug to treat skin inflammation in psoriasis.

The ALADIN score can predict how well patients with alopecia areata will respond to JAK inhibitor treatments.

Cells that move well may improve hair loss treatments by entering hair follicles.

Blocking JAK1 or JAK3 helps reverse hair loss in a mouse model of alopecia areata.

Monilethrix is linked to the type II keratin gene on chromosome 12q13.

A specific gene variant is linked to severe insulin resistance and hormone imbalance in a teenage girl.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

Scarring alopecia involves increased immune cells and specific gene changes near damaged hair follicles.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Higher levels of IL-1a and IL-1RA were found in severe alopecia areata cases.

Blocking IL-17 can reduce joint inflammation in Ross River Virus infections.

Higher levels of certain DNAs in blood may indicate hair follicle damage in alopecia areata patients.

Interleukin-6 may play a role in causing alopecia areata.

Activating TLR3 helps improve skin and hair follicle regeneration after wounds.

Expanded CD8+ T cells are linked to Alopecia Areata and may cause relapse after treatment.

The ovine Hoxc-13 gene is crucial for hair formation and may have other skin functions.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

A unique gene mutation was found in a family with monilethrix.

MEX3A is crucial for maintaining intestinal stem cells in mice.