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TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

A specific RNA helps increase the growth of skin cells in Liaoning cashmere goats by working with a protein to boost a growth-related gene.

TCF/Lef1 is essential for skin barrier function by regulating lipid metabolism.

IL-25 helps heal diabetic wounds by improving blood vessel and skin cell functions.

Certain IL-18 gene variations may increase the risk of alopecia areata.

Overexpression of LRIG3 in skin causes hair loss.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Blocking IL-17 can reduce joint inflammation in Ross River Virus infections.

p120-catenin helps control skin inflammation by regulating cadherin levels.

Certain immune cells, when activated by specific signals, can encourage hair growth.

A(1-7) treatment reduces symptoms of lupus in mice.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

IL-9 increases skin cell movement but decreases their ability to invade, and this effect is controlled by cell contractility, not by MMPs.

Lower GATA3 levels in mice help hair regrow by changing certain immune cells.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

IL-36α helps grow new hair follicles and speeds up wound healing.

Mechanical stress causes ligament thickening through WISP-1 and Hedgehog signaling.

IL-2 treatment causes skin eruptions and other reversible side effects, and may play a role in psoriasis.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

The Itpr3 gene causes a specific hair pattern in mice.

Removing Ctip2 in skin cells causes skin inflammation similar to atopic dermatitis.

Certain genetic variations in IL-16 may increase the risk of alopecia areata.

miR‑339‑5p can slow down hair follicle stem cell differentiation by targeting DLX5.

ILK is essential for skin development, pigmentation, and healing.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Patients with Alopecia Areata have higher levels of certain inflammatory markers.

HuR is essential for Treg function and preventing autoimmunity.

MC-1R is present in skin cells and may help reduce inflammation.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.