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The first Japanese case of a genetic hair disorder caused by specific mutations in the LIPH gene was identified.

HLA can be linked to autoimmune hepatitis.

Dupilumab effectively treats severe skin issues in a rare genetic disorder.

The enzyme for activating vitamin D is found in many body tissues, not just the kidneys.

Targeting PTEN can improve healing in venous leg ulcers.

The STIM1 R304W mutation in mice leads to bone changes and teeth hair growth.

High levels of the protein Flightless I worsen ulcerative colitis symptoms in mice.

Topical IKKα inhibitors may help prevent CCS tumours.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

Certain proteins involved in DNA modification may affect the genetic changes in systemic lupus erythematosus and could indicate the disease's activity.

Belimumab reduces disease activity, steroid use, and flares in lupus patients.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.

A faulty KLHL24 gene leads to hair loss by damaging hair follicle stem cells.

Certain gene mutations in Japanese people are linked to different types of hair loss, with some causing mild hair thinning and others leading to complete baldness.

A new mutation in the hHb1 keratin gene is linked to the hair disorder monilethrix.

CXCL12+ fibroblasts help recruit neutrophils to fight skin infections.

A mutation in the IL2RA gene increases the risk of alopecia areata.

Alpha 6 + /MHCI - cells have stem cell traits and are similar to mouse hair follicle stem cells.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

The keratin 19 promoter effectively targets genes in specific epithelial cells in mice.

Three specific mutations in the LIPH gene can cause hair loss by damaging the protein's structure and function.

Two new gene mutations cause a rare hair disorder.

TCF/Lef1 is essential for skin barrier function by regulating lipid metabolism.

Anifrolumab may help improve symptoms in patients with overlapping autoimmune diseases.

The protein FLCN is involved in cellular cleanup and is regulated by ULK1.

Newborns with the common rash Erythema Toxicum have many active mast cells in their skin, but these cells don't produce the LL-37 peptide.

The E2 protein affects gene activity in hair follicles of mice.