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Dimethyl fumarate speeds up wound healing in IL-36Ra deficient mice by reducing NET formation and oxidative stress.

IL-36α helps grow new hair follicles and speeds up wound healing.

IL-1 family cytokines are crucial for skin defense and healing, but their imbalance can cause skin diseases.

A protein called IL-36γ causes skin side effects from certain cancer treatments when combined with a common skin bacteria.

Alarmin cytokines are key in controlling skin immunity and inflammation.

Understanding molecular mediators can improve skin healing treatments.

Hidradenitis suppurativa is linked to autoinflammation and immune system issues.

Understanding biological mediators in skin healing can improve treatments for skin wounds.

Keratinocytes contribute to hidradenitis suppurativa by causing inflammation and worsening the condition.

Biologic agents are effective for severe hidradenitis suppurativa when other treatments fail.

Fat, bone, and the brain are interconnected in regulating energy and health.

Secukinumab and acitretin together may quickly and effectively treat severe generalized pustular psoriasis.

Engineered cytokines show promise for improving tissue healing and safety in regenerative medicine.

IL-17-producing γδ T cells help improve bone healing.

Anti-IL-12/IL-23 antibody therapy effectively treats psoriasiform skin lesions in IBD patients.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

IL-6 contributes to hair loss by shortening the growth phase and causing hair follicles to regress.

Targeting the IL-23/IL-17 pathway effectively treats several inflammatory skin diseases.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

UVB exposure in human skin causes macrophages to produce more IL-10 and less IL-12, leading to immunosuppression.

Blocking IL-12/IL-23p40 helped reverse severe hair loss in patients.

Blocking IL-17 signaling may reduce skin inflammation and delay aging.

Netherton syndrome has two subtypes with shared immune traits but different allergic and immune responses, suggesting targeted treatments.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Equisetum debile extract, especially the ethyl acetate type, may be a promising natural ingredient for anti-hair loss products.

IL-15 is key for T cell function and could help improve treatments for immune-related diseases.

IL-1 promotes fat cell growth in skin, while WNT inhibits it and encourages scar formation.

IL-4 gene variation may increase the risk of alopecia areata in Turkish people.

IL-3 worsens lupus nephritis and blocking it improves kidney health.