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Junctional proteins stabilize the inner root sheath and connect the companion layer in human hair.

Chitosan/LiCl composite scaffolds help heal deep skin wounds better.

Certain immune markers may predict chemotherapy response in mesothelioma, and nivolumab is a tolerable and effective treatment for advanced non-small cell lung cancer.

Mesenchymal stem cells help heal wounds by using Cx43 hemichannels to improve tissue repair.

A patient with a PLEC mutation has epidermolysis bullosa, muscular dystrophy, and myasthenia gravis, which improved with steroid treatment.

848 genes related to fat and metabolism are less active in people with Central Centrifugal Cicatricial Alopecia.

Certain genetic changes in the LSS gene cause a rare skin and hair condition.

Higher levels of certain immune cells and proteins are linked to more severe lupus symptoms.

Nε-(carboxymethyl) lysine delays hair growth by blocking a key protein.

Blocking CISD1 reduces hearing loss from cisplatin in mice.

Interleukin-2 treatment improved hair growth, sexual function, and reduced fungal infection in a patient with thymoma-related symptoms.

Hair follicles help attract immune cells to minor skin injuries.

A combination of ixekizumab and tofacitinib successfully treated severe scalp cellulitis, leading to hair regrowth.

Lack of functional CYLD in mice leads to early aging and cancer.

Dual TCR Treg cells are common in mouse tissues and vary by location.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

Mechanical stress causes ligament thickening through WISP-1 and Hedgehog signaling.

The IRF2BP2 gene affects sheep fleece quality by influencing fiber traits.

FCER1A and RGS1 may help diagnose and treat systemic lupus erythematosus.

Neutrophils quickly respond to skin injury.

Different sPLA2 enzymes have unique roles in phospholipid metabolism and biological processes.

The method shows how hair lipids form specific patterns and their roles in hair structure.

Mutations in iRhom2 affect hair and skin in mice and are linked to esophageal cancer, with ADAM17 playing a crucial role.

CXXC5 can both suppress and promote cancer, making it a complex target for treatment.

Low serum complement levels in SLE patients don't always match with disease flares; monitoring C3 and C4 is useful, but cell-bound complement products might better indicate disease activity.

Interleukin 6 may help protect skin without causing inflammation.

Ustekinumab treatment may reveal a genetic tendency for steatocystoma multiplex in psoriasis patients.