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Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

iNKT cells help develop and maintain healthy skin in young mice.

IL-18 signaling helps mature Tregs move into the thymus.

Skin grafts on mice can cause an immune response leading to hair loss, useful for studying human hair loss conditions.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

Applying a DNA vaccine to skin with active hair growth boosts immune response and protection against anthrax in mice.

Intensive immunosuppression can reduce relapse rates in multiple sclerosis patients, despite some side effects.

Early over-expression of FoxN1 harms immune and skin development.

Different types of atopic dermatitis were linked to specific genetic and immune changes, suggesting that severe cases might need stronger immune-targeting treatments.

PINK1 is important for controlling gut immune responses linked to early Parkinson's disease.

CD8+ T cells drive alopecia areata, while regulatory T cells are protective.

Certain immune system genes are linked to a higher risk of psoriasis and psoriatic arthritis, while others may offer protection.

Vitiligo and alopecia areata may have similar causes despite their differences.

Rats can develop an immune response to prion protein peptides, but it may cause severe skin issues in older rats.

The patient had a severe allergic reaction to gold treatment.

Melanocytes might be targeted by the immune system in people with alopecia areata, but more research is needed.

Recent selection on immune response genes was identified across seven ethnicities.

FoxN1 overexpression in young mice harms immune cell and skin development.

Vaccines work well in Netherton syndrome patients, similar to healthy people.

The immune system can cause permanent skin and hair whitening by attacking pigment cells.

A specific genetic change is linked to mental disorders, intellectual disability, and possibly autoimmune disease in a family.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Immunocompromised patients can develop skin and hair issues due to a virus.

A boy with severe immune deficiency and Epstein-Barr virus died from high-grade B-cell lymphoma.

Chronic graft-versus-host disease in the skin shows strong Th1 immune response and unique barrier issues.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.