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Oxidized LDL reduces cell growth but affects stem cell differentiation less negatively than cytokine-induced inflammation.

Local inflammation worsens autoimmune skin conditions by increasing antibody buildup.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

Using fat-derived stem cells with the drug meglumine antimoniate can help control skin disease and reduce parasites in mice with leishmaniasis.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

The CD4 protein may play a role in the behavior of certain skin cells, affecting their growth, movement, and differentiation.

Transplant success has improved with better immunosuppressive drugs and donor matching.

Interferon, especially alfa interferon, is an effective treatment for cutaneous T-cell lymphoma with manageable side effects.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

IL-17-producing γδ T cells help improve bone healing.

CD80 on skin stem cells helps expand Treg cells to aid wound healing.

Removing certain hair follicle stem cells worsens skin reactions to allergens.

The research developed methods to test drugs that could protect and restore hair follicle protection in a hair loss condition.

EGFRi/MEKi treatments cause hair follicles to lose some immune protection, leading to inflammation.

AD-derived keratinocytes effectively mimic inflammation in atopic dermatitis.

Megestrol acetate helps fat-derived stem cells grow, move, and turn into fat cells through a specific receptor.

Stromal cells in melanoma promote tumor growth and spread.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

γδ T cells adapt uniquely to different tissues in mice.

Lysophosphatidic acid boosts stem cell growth and movement by creating reactive oxygen species.

ADM scaffolds help skin heal by promoting a healing-type immune response.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Restoring nerve-macrophage communication may help treat autoimmune diseases.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

Targeting MIG and MCP-1 may help treat inflammation in alopecia areata.

HDAC inhibitors help brain cells grow and improve brain function.