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Sensory neuron and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Sensory neuron and Merkel cell changes in the skin happen independently during normal skin maintenance.

Lrig1 marks a unique group of stem cells in mouse skin that can become different skin cell types.

Sensory neuron changes and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Sensory neurons and Merkel cells remodel at different rates during normal skin maintenance.

Sensory neuron remodeling and Merkel-cell changes in the skin happen independently.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Serine is vital for hair follicle stem cells to balance hair growth and skin repair.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

Hair follicle stem cells help maintain skin health and could improve skin replacement therapies.

Current methods can't fully recreate skin and its features, and more research is needed for clinical use.

Researchers found a new gene, hacl-1, that is active in mouse hair follicles during hair growth and may be important for hair biology.

Mice without certain skin proteins had abnormal skin and hair development.

Different types of stem cells in hair follicles play unique roles in wound healing and hair growth, with some stem cells not originating from existing hair follicles but from non-hair follicle cells. WNT signaling and the Lhx2 factor are key in creating new hair follicles.

Biomaterials can help reduce skin scarring and improve wound healing.

Mechanical stimulation and new therapies show promise for hair regrowth.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Caspase-1 helps hair stem cells move to heal wounded or inflamed skin.

Stem cells in hair follicles are diverse and change throughout the hair cycle.

Immune-epithelial interactions are crucial for tissue repair, but unchecked can cause diseases.

Activin B helps start and grow hair follicles in mice.

Skin-derived stem cells show promise for improving wound healing and creating transplantable tissue.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

Sweat gland stem cells help maintain glands, aid wound healing, and can regenerate skin structures.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

Blocking the Wnt pathway could lead to new treatments for cancer and tissue repair but requires careful development to avoid side effects.

Collagen XVIIα1 decline causes skin and hair aging.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.