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Two new IRAK4-inhibitors effectively reduced skin inflammation and immune response markers in healthy volunteers.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

Dimethyl fumarate speeds up wound healing in IL-36Ra deficient mice by reducing NET formation and oxidative stress.

IL-9/IL-9R signaling can negatively affect human hair growth and may be a target for treating hair loss conditions.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Keratin 6A increases skin inflammation, suggesting it could be a target for treating certain skin diseases.

Higher levels of IL-1a and IL-1RA were found in severe alopecia areata cases.

Certain immune markers may predict chemotherapy response in mesothelioma, and nivolumab is a tolerable and effective treatment for advanced non-small cell lung cancer.

TEC kinases may help cause inflammation in vitiligo and could be targeted for treatment.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Laminin-511 is involved in psoriasis development and can be regulated by cannabinoid receptor type 1.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

The ID2 gene can help distinguish between sensitizers and irritants in skin cells.

Different γδ T cell types have unique roles in causing alopecia areata.

Only one of the two K16 genes on chromosome 17 makes a functional protein for keratin filaments.

ILC1-like cells can independently cause alopecia areata.

Keratin 17 is crucial for cell growth in wound healing by aiding protein synthesis.

Treatment with biologic agents can significantly improve psoriasis symptoms, and blood biomarkers could potentially predict individual patient's response to treatment.

Proinflammatory fibroblasts and vascular endothelial cells are key in keloid development.

Higher levels of MIG and IP-10 may help diagnose and monitor Alopecia Areata.

Blocking IL-8 can reduce skin rashes from cancer treatment.

SIG-1451 could be a promising new treatment for atopic dermatitis.

IL-4 and IL-13 might play a role in a type of hair loss similar to alopecia areata.

ILC1-like cells may contribute to hair loss in alopecia areata.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.