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Differences in androgen receptor expression and tissue properties may lead to higher cryptorchidism risk in certain rats.

Dihydrotestosterone increases the activity of an enzyme in pubic skin cells that converts testosterone to dihydrotestosterone.

Nuclear AR levels give better insight into hormone effects in skin conditions.

ROS may affect hair loss related to hormones.

Androgens like testosterone affect hair growth and oil production differently across body parts and individuals.

Therapies targeting the androgen receptor are being developed to treat prostate cancer and improve bone and muscle health.

Androgens like testosterone increase colon muscle contractions by affecting calcium pathways.

Androgen receptors help prostate cancer cells grow and resist drugs.

The study concludes that a genetic mutation in TFM mice leads to reduced androgen receptor activity, affecting the body's response to male hormones.

Leydig cells can cause testosterone-secreting adrenal tumors in women.

Testosterone is important for male sexual traits, reproduction, muscle and bone health, blood production, and metabolism, and works both directly and after being changed into other hormones.

Androgens reduce THY1 in skin cells, leading to less fat, more fibrosis, and worse healing in males.

Mutations in the androgen receptor gene cause different levels of androgen insensitivity, making it hard to create simple tests for the condition.

High-dose androgen therapy can help manage certain types of androgen resistance.

Male rats have androgen receptors in their liver, which are different from proteins that bind estrogen.

Estrogen affects how vitamin A is processed in mouse skin, which may impact acne treatment, hair growth, and skin defense.

Too much AKR1C3 enzyme causes resistance to finasteride by increasing testosterone.

Androstanediol glucuronide is a reliable marker for hirsutism in women.

Fibroblasts help understand androgen resistance at the cellular level.

Male hormones promote hair cell growth by using a growth factor from nearby skin cells.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

New compounds were made that are promising for prostate cancer therapy.

Higher androgen levels and site-specific AR expression cause sex-related skeletal differences, and certain steroids can boost AR expression and androgen effects in bone cells.

Insufficient androgen action in male fetuses can cause genital development issues due to genetic mutations or environmental chemicals.

Antiandrogens have important biological effects, but more research is needed to understand them fully and compare their effectiveness and side effects to other treatments.

Canine hair follicle cells metabolize DHEA and testosterone differently than human cells.

Certain changes to the B-ring of androgen receptor ligands can increase their effectiveness for potential treatments of muscle and bone conditions.