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Promising treatments for EBS include anti-inflammatory drugs, antibiotics, creams, mTOR inhibitors, and gene editing, but more trials are needed.

Tiny infundibular cysts are the main lesions in chloracne, not comedones.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

Paneth cells help support stem cells and aid tissue regeneration after injury.

Temporary activation of β-catenin can create new hair follicles, but ongoing activation is needed to keep hair follicle tumors.

A new one-step test can quickly identify skin cancer during surgery.

Ber‐EP4 marks cells related to the secondary hair germ in hair follicles.

Type 1/17 inflammation in psoriasis increases skin cell growth due to a molecule that could be a new treatment target.

Keratin mutations cause skin diseases and could lead to new treatments.

Sub3 is essential for fungus adherence but not for skin invasion.

QLT0267 stops hair follicle cell growth and movement.

LRIG1 is linked to better survival in Merkel cell carcinoma.

The research confirms that Hidradenitis Suppurativa is characterized by increased inflammation, disrupted skin cell organization, and abnormal metabolic processes.

The study found that a key immune pathway protecting hair follicles is reduced in a mouse model of scarring hair loss.

Porcine and human pilosebaceous units are very similar.

Skin cells release substances important for healing and fighting infection, and understanding these could improve skin disorder treatments.

Skin bacteria can help wound healing by activating certain immune cells.

Acne patients' skin, both with and without lesions, shows a strong immune response and higher antimicrobial activity.

iPSC-derived stem cells on a special membrane can help repair full-thickness skin defects.

Hidradenitis suppurativa may be a type of autoinflammatory skin disease linked to gene mutations and immune system issues.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

The method and source of keratinocytes affect the structure of reconstructed skin.

EGFRi/MEKi treatments cause hair follicles to lose some immune protection, leading to inflammation.

The cornea develops independently of the lens, following its own default pathway.

The new protocol using Cellutome™ and RCM safely assesses wound healing in detail.

Live imaging helps us understand skin immune responses and develop treatments.