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Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

The SLICK1 allele in Holstein heifers affects hair and immune traits without altering prolactin signaling.

Activin A increases inner ear hair cell development, while follistatin decreases it.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

Proper niche formation in Drosophila requires Slit-Robo signaling for cell migration.

Wound healing involves complex signaling that stops bleeding, reduces damage, and repairs skin, sometimes without scarring.

Blocking hair follicle development stops key gene signals needed for hair growth in mice.

The Wnt signaling pathway is crucial for regeneration and could help advance human medicine.

Phospholipase C-δ1 is crucial for normal hair development.

Removing UBE2N from skin cells causes inflammation and immune response, which can be lessened with specific inhibitors.

A specific signal from hair cells controls the tightening of the surrounding muscle, which is necessary for hair shedding.

TTNPB helps turn stem cells into neural stem cells, improving depression-like behaviors in rats.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.

CLK1 is needed for skin cells to become epidermal cells but not sebocytes.

HOXC13 is essential for hair and nail development by regulating Foxn1.

ACKR2 helps prevent skin scarring and hair loss by controlling inflammation.

A20 protein is crucial for normal skin and hair development.

CXXC5 is a protein that controls cell growth and healing processes, and changes in its activity can lead to diseases like cancer and hair loss.

Weak cell junctions disrupt hair follicle stem cell rest.

Covering a wound can stop hair growth by promoting scarring, but boosting a process called Wnt signaling can help hair grow back even when the wound is covered.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Wnt signaling is crucial for bone regeneration.

Tofacitinib helps mice grow more hair by increasing noggin and BMP4 levels, possibly better than minoxidil.

NFATc1 is crucial for keeping hair follicle stem cells inactive.

β-Catenin signaling is involved in brain cell growth after injury and could be a therapy target.

SPT6 prevents excessive skin inflammation by blocking a feedback loop.

Zebrafish need MYC and FGF to regenerate inner ear hair cells.

EGFR helps mouse hair follicles stop growing by reducing certain growth regulators.