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15d‐PGJ2 cream protects against UVB-induced skin damage in mice.

Folliculin deficiency causes problems with cell division and positioning due to disrupted RhoA signaling and interaction with p0071.

Microfluidic models improve testing for aging, wound healing, and oral tissue, reducing animal testing.

Bifidobacterium animalis subsp. lactis J12 helps reduce atopic dermatitis symptoms.

HPV8 E6 gene causes growth of certain skin stem cells.

Inherited color dilution in rabbits is linked to DNA methylation changes.

Wharton's jelly stem cells show diverse traits and functions.

Oral JAK inhibitors improved alopecia areata by modulating immune responses and boosting hair growth.

ocu-miR-205 affects hair density in Rex rabbits by altering hair follicle growth and signaling pathways.

Nε-(carboxymethyl) lysine delays hair growth by blocking a key protein.

Nexin 1 helps control hair growth in young rats.

JAK inhibitors can be safely used in alopecia areata patients with latent hepatitis B or tuberculosis, with proper monitoring.

Longdan Xiegan decoction is more effective than conventional medicine for treating eczema.

PRX01, PRX44, and PRX73 affect root hair growth by interacting with extensins in Arabidopsis.

Early diagnosis, stopping the suspected drug, and supportive care are crucial for treating Stevens-Johnson Syndrome and Toxic Epidermal Necrolysis.

A deletion in the CDH3 gene causes a rare disorder with short hair and vision loss.

The LustrivaTM BH-002 supplement and hair serum improved women's hair, skin, and nail health and appearance.

Upadacitinib effectively treats pyoderma gangrenosum.

Genetic screening for NUDT15 polymorphisms is crucial for patients taking azathioprine, especially in Asians.

Stopping and restarting JAK inhibitors during pregnancy in women with alopecia areata leads to hair loss and regrowth, with most pregnancies resulting in healthy babies.

Tofacitinib helped a 19-year-old regrow hair after other treatments failed.

Mouse Scube3 affects teeth, tongue, vibrissae, and eye development, but not facial structure or limb growth.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

A genetic duplication on chromosome 5 was linked to a woman's unique combination of medical conditions.