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A genetic mutation in the SGK3 gene causes hairlessness in Scottish Deerhounds and may relate to human hair loss.

A gene called Gk5 controls lipid production in the skin and affects hair growth.

Spiny mice have resilient, large mitochondria that help them regenerate tissue.

Smad2 and Smad3 are essential for normal skin development, and their absence causes severe skin issues and cancer.

SFRP2 boosts Wnt3a/β-catenin signals in hair growth cells, with stronger effects in beard cells than scalp cells.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

Hearing decline in SAMP8 mice starts before outer hair cell loss and may be linked to other changes.

A gene mutation causes curly hair and hair loss in rats.

TSC2 is crucial for proper hair follicle development and patterning.

Finasteride, at doses of 5mg or higher, may negatively affect male fertility by reducing the expression of certain genes involved in sperm production.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Genetically modified cells can regenerate skin and hair in rats.

Lack of TrkC receptor delays hair follicle development.

SMAD4 is crucial for muscle repair in young adults but not in aged mice.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

Adult mice with CBS deficiency show minimal health issues and normal lifespan despite high homocysteine levels.

The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

Overexpression of a specific receptor in mice skin causes skin thinning, early skin barrier formation, eye issues, and hair loss.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Progranulin overexpression leads to shorter, thinner hair and increased cell death in mouse hair follicles.

High levels of ST14 and TMEFF1 proteins in ovarian cancer are linked to worse patient outcomes and may be a new treatment target.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

SLICK cattle have better heat tolerance due to specific gene expression and pathway differences.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

The Oat mouse model shows mild retinal degeneration, useful for testing treatments.