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Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

Smad2/3-dependent TGF-β signaling increases during wound healing.

B6.Cg-Tyr c−2J Hr hr /J mice have a stronger delayed sunburn reaction and are good for UV research.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.

Altering the keratin 17 gene in mice hair follicles caused temporary hair issues, but changes were minimal and short-lived.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

TCDD changes skin cell growth and keratin production in mice.

The PML protein helps prevent skin cancer in mice.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

The improved genome of the African spiny mouse helps study its tissue regeneration.

The technique successfully promoted hair growth and skin renewal in mice.

Polyamines are crucial for skin tumor development, and inhibiting them can prevent tumors.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.

The research identified new skin traits in mice, some linked to human skin conditions.

Inactivating both p53 and Rb genes in mice speeds up aggressive skin cancer development.

Hair loss in certain mice is linked to changes in keratin-related genes.

Female mice with disrupted 5α-reductase 1 had significant metabolic issues, including stress response problems, insulin resistance, liver fat buildup, and obesity.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

Researchers successfully transplanted hair follicles in mice, which survived well and helped in wound healing.

SGK3 is essential for proper hair growth and health.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Two-photon microscopy effectively tracks live stem cell activity in mouse skin with minimal harm and clear images.

The B2C promoter works in sheep cells but not in mouse embryos.

Super-enhancers control CD25 expression in specific cell types, affecting immune function.

Removing Gsdma1/2/3 genes reduces skin cell overgrowth by blocking a specific cell pathway.

Overexpressing the Tβ4 gene in goats can increase cashmere production.

A PIK3CA mutation in Schwann cells causes severe nerve damage and increased glycolysis, but early treatment can help.

A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

Deleting Snai2 and Snai3 causes fatal autoimmunity.