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A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

A new keratin gene was found in mice, explaining hair growth.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.

Zfp157 is active in many mouse tissues during development and in specific adult cells.

Increasing SSAT makes skin more prone to cancer.

Over-expression of Sp2 can lead to cancer by preventing proper stem cell differentiation.

Activating Tgr5 may help treat hair loss and bone loss.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

A new mouse model for vitiligo helps study immune responses and potential treatments.

cgVEGF164 boosts hair follicle growth in mice.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.

ErbB2 signaling is crucial for skin cell growth and cancer development in mice.

Dimethylarsinic acid speeds up skin tumor growth in certain mice.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

The UHS promoter is specific to mouse hair follicles.

The vector successfully directed specific gene expression in hair follicles.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

The mouse model helps study and develop treatments for radiation-induced saliva reduction.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Akt2 and SGK3 are both important for normal hair growth and development.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.