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Three molecular subtypes of advanced skin T-cell lymphoma were identified, with potential biomarkers for predicting treatment response and disease progression.

BMP signaling is essential for the development of touch domes.

Stat3 affects skin cancer development by altering keratinocyte stem cell behavior.

Prompt treatment of inflammatory tinea capitis (kerion) is crucial to prevent scarring and hair loss.

Sensory neuron and Merkel cell changes in the skin happen independently during normal skin maintenance.

ORS and hair matrix cells balance growth and differentiation better than normal keratinocytes, with human dermal fibroblasts crucial for proper differentiation.

Altering the keratin 17 gene in mice hair follicles caused temporary hair issues, but changes were minimal and short-lived.

BSSP may help skin tumors grow and could be a marker or target for skin cancer treatment.

A mutation in the FAM83G gene is linked to skin and hair abnormalities in two related individuals.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

Keratoacanthoma and some squamous cell carcinomas are linked to hair follicles, while others are not.

Integrin-linked kinase is crucial for melanoblasts to properly colonize the skin.

Different types of stem cells in the skin contribute to the variety of melanoma forms.

Keratoacanthomas on lips may originate differently than those on skin.

Krtap11-1 is important for hair strength and structure.

Scientists found cells in hair that are key for growth and color.

Trichofolliculoma has specific cytokeratin patterns that help in its diagnosis.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Melanocyte activity in hair follicles is linked to the hair growth cycle, being active in growth phases and inactive in rest phases.

New hair regrowth model proposed, imiquimod found to kill skin cancer cells, T-cadherin loss linked to invasive skin cancer, no clear gene link to skin cancer after transplant, and study on children's hair loss shows male dominance and genetic ties.

The lump on a woman's scalp was a rare, potentially cancerous tumor from the hair follicle, not a common cyst.

KRT72 gene helps form hair.

A rare scalp infection in a child developed into a kerion with additional skin symptoms.

Too much IKZF1 and Ikaros protein may cause alopecia areata.

KRTAP2-3 could help predict cancer recurrence by identifying specific cancer cells.

Scientists used stem cells to create a model of the skin disease Epidermolysis Bullosa simplex, which helped them understand its molecular mechanisms and could aid in finding treatments.

Nε-(carboxymethyl) lysine delays hair growth by blocking a key protein.