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The keratin 5 mutation in a family with epidermolysis bullosa simplex was due to mosaicism, not a new mutation.

Activating Kras in mouse skin causes excess skin and hair loss.

Lgr5+ stem cells do not cause skin tumors.

Keloid scars may form due to changes in skin cell characteristics and specific protein signaling.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

Nestin-expressing progenitor cells become outer root sheath keratinocytes.

BRG1 is essential for skin cells to move and heal wounds properly.

Epidermal cysts come from the skin, while pilar cysts come from hair follicles.

Merkel cells develop independently of nerves and are linked to specific hair follicles in mice.

Terbinafine effectively treated kerion celsi despite disrupted immune responses.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Keratin-75 is secreted by ameloblasts in a unique way without a signal peptide.

Some canine hair follicle tumors contain amyloid deposits, with a protein called CK5 involved in their formation.

Keratin mutations may cause scarring alopecia by damaging hair structure.

Trichocytic cytokeratins are found in hair, nails, tongue, and thymus cells, showing complex regulation in tissue development.

The cuticle layer in hair follicles thickens during keratinization due to incomplete cytosol loss.

Stem cell behavior varies with stimuli, and lineage changes can happen without affecting stem cell division.

Seborrheic keratoses may partly come from hair follicle cells.

YAP and TAZ proteins control skin cell growth and repair.

A mutation in the KRT71 gene causes a hair disorder by disrupting hair follicle structure and texture.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

EGFR deficiency in skin causes hair follicle issues and inflammation.

Trichoblastic carcinoma may be a distinct type of skin cancer different from basal cell carcinoma.

The antibody created from BCC tissues reacts similarly to both BCC and hair follicles, suggesting BCC may come from hair follicle cells.

Keratin mutations cause skin diseases and could lead to new treatments.