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Overexpressing PKCα in mice skin increases inflammation but doesn't affect tumor growth.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

Phosphatidylinositol 4-kinases are crucial for root growth, defense, and immunity in Arabidopsis thaliana.

SPRY1 deficiency in skin cells causes stem cells to move to the skin surface, leading to increased pigmentation.

CaBP1 and CaBP2 are important for continuous hearing by preventing inactivation of calcium currents in ear cells, with CaBP2 also able to restore hearing when reintroduced.

Reducing SFRP1 can promote hair growth and may help treat hair loss.

Deleting Gpr54 speeds up hair growth and regeneration.

CD271 is crucial for maintaining healthy skin and preventing inflammation.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

Acid can block TRPV3 from outside the cell but boost its function from inside.

A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

Alk1 in specific cells is crucial for proper nerve branching and hair function.

The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

mEphA1 receptor tyrosine kinase is important for skin and hair development and may play a role in certain diseases.

CREB, a protein that can promote cancer traits, is controlled by β-catenin in skin cancer cells.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

TRPS1 loss in balding scalp areas contributes to hair loss in androgenic alopecia.

KRT72 gene helps form hair.

CaBP1 and 2 are important for maintaining the activity of calcium channels necessary for hearing in inner ear cells.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

Mouse keratin 6 genes evolved independently from human ones and are regulated differently.

Keratin K6irs is a marker for the inner root sheath of hair follicles in mice and humans.

Blocking certain proteins in mouse skin can reduce and shrink skin tumors.

Keratin 17 is crucial for cell growth in wound healing by aiding protein synthesis.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

A specific DNA region controls skin cell gene expression by working with certain proteins.

The 190-kbp domain contains all human type I hair keratin genes, showing their organization and evolution.

TRPV3 and TRPV6 channels change structure to regulate calcium and heat responses.

The Celsr1 gene is crucial for normal hair patterning in mice.