Search

everythinglearnresearchcommunity

for

Search:↓

The research cloned keratin 7 genes from humans, mice, and marsupials, found similarities between human and mouse genes, and discovered new areas of K7 expression in mice.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

New mouse models help study melanocytic cells for melanoma research.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Tsc2-deficient stem cells can help understand and treat TSC-related tumors.

Different keratins have unique expression patterns in mouse skin cells.

Removing both KLK5 and KLK7 proteins can prevent death and skin issues in Netherton syndrome.

TCDD changes skin cell growth and keratin production in mice.

Akt2 protein is essential for normal cell division in early mouse embryos.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

CTIP2 may help in skin development and maintenance.

A new mouse model helps study melanocyte cells using GFP expression.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Cathepsin L deficiency causes large, abnormal cell structures and health issues in mice.

Mice with human skin protein K8 had more skin problems and cancer.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

Mutations in iRhom2 affect hair and skin in mice and are linked to esophageal cancer, with ADAM17 playing a crucial role.

Tortoiseshell tomcats with XX/XY chimerism can be fertile.

Akt2 and SGK3 are both important for normal hair growth and development.

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

A specific hair protein variant increases the spread of breast cancer and is linked to worse survival rates.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

A new gene mutation may allow some piebaldism patients to regain skin color in white patches.

Lack of functional CYLD in mice leads to early aging and cancer.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

Keratin2-6g is crucial for hair follicle development, with mutations causing cell degeneration and vacuolation.