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Kojyl cinnamate ester derivatives can promote hair growth by increasing adiponectin production in fat tissues.

KID syndrome should be reclassified as an ectodermal dysplasia.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

XEDAR triggers a specific signaling pathway in cells.

A new mutation in the XEDAR gene might cause a rare skin condition called hypohidrotic ectodermal dysplasia.

CREB, a protein that can promote cancer traits, is controlled by β-catenin in skin cancer cells.

CPK1 helps root hair growth in Arabidopsis by activating channels for calcium signaling.

KGF-1 135 is a stable and effective alternative for treating oral mucositis.

The EDA pathway plays a key role in bone development by interacting with other signaling pathways.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

A new EDA gene variant causes X-linked hypohidrotic ectodermal dysplasia in a Chinese family.

PEG and keratin scaffolds can effectively deliver protein drugs by controlling release based on pH levels.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

Chemokine signaling is important for hair development.

c-Kit is important for heart regeneration and cancer development.

XEDAR deficiency prevents muscle degeneration in EDA-A2 transgenic mice.

Researchers found a new mutation in the FERMT1 gene in a Spanish family with Kindler syndrome.

The new model helps understand and develop treatments for genetic skin disorders like AEC.

A cat's poor wound healing was linked to a genetic deletion in the COL5A1 gene.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

A PKP1 gene mutation causes skin fragility and hair loss in Chesapeake Bay retriever puppies.

EDA signaling is linked to skin disorders, various cancers, and liver disease.

A new mouse mutation causes skin and hair defects due to a gene change.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Ectodysplasin inhibits Wnt signaling to help form hair follicles.

CLK1 is needed for skin cells to become epidermal cells but not sebocytes.

A new EDA gene mutation was found in a Chinese family with a specific skin disorder.

A rabbit gene important for hair development was identified and detailed.