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ErbB2 signaling is crucial for skin cell growth and cancer development in mice.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Cetaceans lost hair genes to adapt to water.

Human hair follicles grown in vitro maintain normal keratin patterns and structure.

A non-toxic formula using polycarboxylic acids strengthens and improves hair.

The malignant pilomatricoma showed strong epithelial keratin expression, suggesting it may not calcify.

Mutations in the Whn gene affect hair keratin gene expression differently.

Nail-matrical fibroblasts can make non-nail cells produce hard keratin, useful for nail repair.

Sheet formation is key to macrofibril structure differences in wool.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

Advancements in hair keratin research could lead to better hair health treatments.

Monilethrix is linked to the type II keratin gene on chromosome 12q13.

Most mouse hair keratin genes are on chromosomes 11 and 15.

Lizard claws have hair-like keratins similar to those in mammals.

LHTric-1 is a specific antibody useful for studying hair and nail formation.

Human hair keratin genes are similar to mouse genes and are specifically expressed in hair follicles.

Lab-grown nail cells show characteristics similar to natural nail and hair.

Brazilian keratin treatment can cause severe skin reactions.

An apple-based supplement was found to stimulate hair protein production, which may help with hair growth.

Enzymatic phosphorylation of hair keratin improves the effectiveness of hair products with cationic ingredients.

The method effectively analyzes human hair proteins, especially nonfilamentous ones.

Epidermal cysts come from the skin, while pilar cysts come from hair follicles.

Hair proteins change location and structure as hair cells mature.

Nucleophilic reagents break down hair keratin, forming more lanthionine and lysinoalanine than in wool.

Hard α-keratin has a universal molecular structure with a specific superlattice arrangement.

The NF-κB effector p65/RelA activates hair keratin genes, aiding hair formation.