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Mutations at Val-889 and Arg-752 disrupt key interactions in the androgen receptor, affecting its function.

Different parts of the nail express different keratins, showing unique patterns of differentiation.

The Glu413Lys mutation in keratin affects hair stability, while Glu413Asp does not.

A KLK5 inhibitor effectively improved skin symptoms in a mouse model of Netherton Syndrome.

Astrotactin2 affects hair follicle orientation and skin cell polarity.

Keratin-associated proteins are part of the developing hair fiber cuticle.

KRT2.13 gene is similar to KRT2.9 but not expressed in hair follicles.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

Understanding proteins in skin structures like claws and hair is crucial for future research.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

Certain skin proteins can form anchoring structures without the protein AMACO.

Newly designed proteins can effectively degrade specific proteins in cells, offering a potential new therapy method.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Krtap11-1 is important for hair strength and structure.

Keratin structure changes during keratinization, but the exact model remains uncertain.

Repetin is a protein involved in skin and hair development, binding calcium and compensating for other proteins when needed.

Feather patterns are influenced by enhancers and chromatin looping, and the structure of protein complexes important for hair growth has been detailed.

AgK114 protein helps in hamster skin injury recovery.

Trichohyalin helps in hair and skin cell structure and function by binding calcium and linking proteins.

Tet1/2/3 enzymes affect hair follicle cell development by influencing BMP signaling.

BKN-1 antibody targets specific keratin in basal cell epithelioma and normal skin basal cells.

The STRIPAK complex is crucial for skin cell organization and creating a functional skin barrier.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

Human hair keratins can form stable nanofiber networks that might help in tissue regeneration.