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Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Blocking BACE1 and BACE2 enzymes causes hair color loss in mice.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Removing certain brain receptors in mice worsens seizure severity and response to treatment during hormone withdrawal.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

Blocking IL-17a can improve age-related smell loss in mice.

Lack of Crif1 in hair follicle stem cells slows down hair growth in mice.

Lef1 is essential for normal skin, hair growth, and healing wounds in mice.

Prolactin may play a significant role in skin and hair health and could be a target for treating skin and hair disorders.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

KLF4 is important for maintaining skin stem cells and helps heal wounds.

Slug (Snai2) helps regulate hair growth timing in mice.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

Dkk4 protein is necessary for the proper development and arrangement of hair follicles.

Knockout mice showed anemia and hair loss, suggesting other ways exist for iron absorption.

EBF1 controls hair type and length.

Sdr16c5 and Sdr16c6 genes regulate a key point in lipid production that affects eye and skin gland function.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Follistatin helps hair growth and cycling, while activin prevents it.

The p53 protein helps control hair follicle shrinking by promoting cell death in mice.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

Gata6 is important for protecting hair growth cells from DNA damage and keeping normal hair growth.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Endothelial TLR2 is crucial for timely wound healing, but HFSC TLR2 is not needed.