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A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Dog skin with hair loss, when transplanted to mice, regrew hair, suggesting the hair loss cause is likely body-wide, not skin-specific.

Overexpressing the Tβ4 gene in goats can increase cashmere production.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

A variant in the KRT31 gene causes a rare hereditary hair disorder called monilethrix.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Fibroblasts from healthy donors can prevent changes seen in recessive epidermolysis bullosa simplex.

Circular RNA ERCC6 helps activate stem cells important for cashmere goat hair growth by interacting with specific molecules in an m6A modification-dependent way.

A new mouse mutation causes skin and hair defects due to a gene change.

Activating Tgr5 may help treat hair loss and bone loss.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.

The humanized AA mouse model is better for testing new alopecia areata treatments.

Activated protein C helps protect mice from long-term radiation damage.

K16 can partially replace K14 but causes hair loss and skin issues.

Lack of functional CYLD in mice leads to early aging and cancer.

Sdr16c5 and Sdr16c6 genes regulate a key point in lipid production that affects eye and skin gland function.

Deleting Gpr54 speeds up hair growth and regeneration.

Erythropoietin overexpression disrupts hair growth and fat formation in mice.

Using a combination of specific cell cycle regulators is better for safely keeping hair root cells alive indefinitely compared to cancer-related methods.

A new mouse model helps understand and find treatments for alopecia areata.

A specific molecular switch, driven by MAPK/ERK signaling, helps spiny mice heal wounds by regenerating skin instead of forming scars.

A bull with a gene mutation was asymptomatic, synthetic retinoids cause hair loss, and new therapeutic targets were identified for skin diseases.

Increasing calcium sensing receptor speeds up skin and hair development in mice.

PrPC is important for neural differentiation in cattle and mouse embryonic stem cells.

ACKR2 helps prevent skin scarring and hair loss by controlling inflammation.

Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

Frizzled 3 and Frizzled 6 together control the orientation of mouse hair follicles.

Activating the Sonic hedgehog gene in mice can start the hair growth phase.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.