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Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

FLCN helps control iron levels in cells.

LRIG1 protein affects hair growth by regulating skin receptors, leading to hair loss when overexpressed.

Removing Lrig1-positive cells in mice leads to temporary loss of sebaceous glands.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

PINK1 is important for controlling gut immune responses linked to early Parkinson's disease.

FA2H is essential for normal fur and sebum production in mice.

A specific gene segment can make mouse skin cells glow, helping study hair growth and gene effects.

Folliculin deficiency causes problems with cell division and positioning due to disrupted RhoA signaling and interaction with p0071.

Hair follicles in mutant mice self-organize into ordered patterns within a week.

Miz1 is essential for proper hair structure and growth.

Progerin affects cell shape but not hair or skin in mice.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.

Mice can correct hair follicle orientation without certain genes, but proper overall alignment needs those genes.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

Deleting the Far2 gene in mice causes sebaceous gland issues and patchy hair loss.

High levels of the protein Flightless I worsen ulcerative colitis symptoms in mice.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Copper treatments increase copper in all tissues, but brindled female mice accumulate much more copper in their kidneys without clinical effects, unlike brindled male mice where brain copper deficiency is clinically significant.

The Shaven mutation in mice affects hair growth and causes a greasy coat due to abnormal lipid content.

Overexpression of certain genes can shorten hair by disrupting the hair-growth cycle.

A mutation in the Sgkl gene causes defective hair growth in mice.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

EBF1 controls hair type and length.

Iron deficiency causes hair loss by affecting hair differentiation and cycling.