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Sensory neuron and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Basal layer skin cells help form the epidermis and hair follicles.

HAP stem cells can repair nerves and spinal cords by becoming Schwann cells.

A small group of slow-growing cells causes basal cell carcinoma to return after treatment.

Certain proteins, Lgr5 and Lgr6, are important markers of adult stem cells and are involved in tissue repair and cancer development.

Nfib in hair follicle stem cells boosts melanocyte stem cell growth and differentiation.

Adding fetal calf serum helps Merkel cells survive and change shape.

Blue-light activation of TrkA improves hair-follicle stem cells' ability to become neurons and glial cells.

A specific group of early-stage melanocytes is reduced in vitiligo-affected skin, which may explain treatment resistance.

The Foxn1(-/-) phenotype disrupts hair growth and affects skin stem cells.

Lymphatic vessels are essential for hair follicle regeneration and growth.

Mesenchymal cells are essential for hair follicle stem cell growth.

Hair follicle stem cells can become nerve cells using specific treatments.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

Primary cilia affect the size and oil production of eye glands but not the oil's makeup.

Sensory neuron changes and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Immune cells are crucial for hair growth and preventing hair loss.

Targeting the HEDGEHOG-GLI1 pathway could help treat keloids.

A rare genetic variant linked to skin cysts was found in blood DNA, suggesting its role in cyst formation.

The research shows that skin cancer likely originates from hair follicles and that certain cell populations expand to promote skin cancer growth.

NGAL indicates abnormal skin cell differentiation.

Lgr5+ stem cells do not cause skin tumors.

Skin-derived precursors in hair follicles come from different origins but function similarly.

Blocking the HEDGEHOG-GLI1 pathway can reduce keloid growth and may be a potential treatment.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Autofluorescence in hair follicle stem cells can interfere with studies but may help isolate these cells.

Human hair cells can change based on their environment, especially interactions with certain skin cells.

Newly divided skin cells quickly move to join skin structures due to tissue tension and specific signals.

Langerhans cells and melanocytes migrate to the skin and hair follicles during early human development.

Hair follicle stem cells can become melanocytes to help treat skin depigmentation.