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Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

PAD3 plays a key role in hair and skin protein structure and may be linked to skin diseases.

Disrupted stem cell signals in hairpoor mice cause hair loss.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

Blocking a specific receptor slows down hair loss in mice.

Hair follicle stem cells in hairpoor mice are disrupted, causing hair loss.

Epiprofin suppresses parathyroid hormone gene activity, helping regulate calcium levels and could be a treatment target for hyperparathyroidism.

Activating TLR3 improves the healing and immune properties of periodontal ligament stem cells.

Gsdma3 affects hair growth by controlling Wnt5a, which influences hair cell development.

Prolactin affects when mice shed and grow hair.

QLT0267 stops hair follicle cell growth and movement.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

Laminin-511 is essential for proper melanocyte movement and development in mice.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

A mouse gene mutation increases the risk of skin cancer.

miR-195-5p reduces hair growth ability in cells by blocking a specific growth signal.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Gasdermin A3 causes hair follicle stem cells to activate too early, leading to hair loss.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Kir6.1 mutations in Cantú syndrome increase channel sensitivity and hyperpolarization, while SUR2B mutations do not.

Removing Lrig1-positive cells in mice leads to temporary loss of sebaceous glands.

Fatty acid transport protein 4 is essential for skin and hair development.

Blocking SEPT4 might help heal wounds and regrow hair faster.

A specific gene mutation causes a rare hair loss condition in a Chinese patient.

Defects in certain proteins cause major heart abnormalities during early development.

Deucravacitinib improves symptoms and reduces inflammation in Lichen Planopilaris.

Early over-expression of FoxN1 harms immune and skin development.

The hairless gene is crucial for hair health, and its mutations cause hair loss.