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Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

The Bsk mutation doesn't involve keratin gene recombination and its cause is unknown.

A new treatment effectively kills antibiotic-resistant bacteria and helps wounds heal faster by boosting the immune response.

Mutations in the FOXN1 gene cause severe immune issues but don't affect hair and nails.

Muse cells from human bone marrow help reduce symptoms of atopic dermatitis in mice.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

Certain enzymes and a growth factor were higher in mice treated with minoxidil or a peppermint and rosemary oil mix, which may be good indicators of hair growth.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Overexpression of LRIG3 in skin causes hair loss.

A new mutation in the mitochondrial DNA was found in a boy with MELAS, even though his family didn't show typical signs.

Yuyi hairless mice lose hair after birth, develop thick, loose skin with folds, and show disorganized skin structure as they age.

Rapamycin and anti-EGFR antibody reduce LAM/TSC cell migration and blood vessel growth in the uterus.

Different sPLA2 enzymes have unique roles in phospholipid metabolism and biological processes.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

A cat's poor wound healing was linked to a genetic deletion in the COL5A1 gene.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Newly designed proteins can effectively degrade specific proteins in cells, offering a promising alternative for targeted protein degradation.

Old C57BL/6 mice with unsynchronized hair cycles show less melanin in their spleens.

Lykoi cats' unique sparse hair is linked to specific genetic variants in the Hairless gene.

WWOX deficiency in mice causes skin and fat tissue problems due to disrupted cell survival signals.

New LSS gene variants help understand congenital hypotrichosis 14 better.

ANE syndrome is caused by a mutation in the RBM28 protein that disrupts ribosome assembly.

Deleting the scd1 gene in mice prevents obesity by disrupting skin lipids and improving heat regulation.

Certain proteins, Lgr5 and Lgr6, are important markers of adult stem cells and are involved in tissue repair and cancer development.

Aged individuals heal wounds less effectively due to specific immune cell issues.

Knockout mice showed anemia and hair loss, suggesting other ways exist for iron absorption.