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Finasteride exposure in pregnancy causes genital abnormalities in male rats.

Finasteride affects male rat genitalia development, causing abnormalities during specific pregnancy days.

Keloids on the penis are rare, often triggered by circumcision, and rarely recur after surgery.

Finasteride significantly reduced sperm production and caused testicular atrophy in hamsters.

Affected males are born with ambiguous genitalia, raised as females, but develop male traits at puberty due to enzyme deficiency.

Finasteride exposure affects gene expression and anogenital distance in male rat fetuses.

Individuals with this condition often develop male traits and identities at puberty despite being raised as females.

Masculinization in affected individuals occurs gradually after puberty due to hormone changes.

Low doses of mixed chemicals cause permanent reproductive malformations in male rats.

Blocking a specific enzyme in male rat fetuses leads to the development of nipples and feminized genitalia.

Testosterone therapy and surgeries like mastectomy improve transgender men's lives and mental health with low risks and high satisfaction.

Male genital development is driven by androgen signaling and understanding it could help address congenital anomalies.

Conditions affecting sex development show that sexual diversity is a natural part of human variation.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

Mutations in the NR3C1 gene cause a rare condition that affects hormone signaling and can lead to various symptoms, with dexamethasone as a treatment option.

Nonclassic 21-hydroxylase deficiency is a common, treatable genetic disorder causing reversible symptoms like acne and hair loss.

Late diagnosis of congenital adrenal hyperplasia can greatly affect physical development, gender identity, and sexual health.

Diagnosing simple-virilizing congenital adrenal hyperplasia can be difficult and requires thorough testing and expert advice.

The patient had complex urinary tract issues and ambiguous genitalia, with physical features suggesting a hormonal imbalance.

Genetic defects in androgen production are linked to male developmental disorders and are improving treatment understanding.

Certain 4-azasteroids are effective at blocking the enzyme that processes testosterone in human skin and could help treat acne, excessive hair growth, and male pattern baldness.

Finasteride shows promise for treating hair loss and excessive hair growth, but more research is needed to confirm its effectiveness and safety.

The first in-prison dermatology clinic in the U.S. faced challenges and found certain skin conditions common among inmates, with rare cases of serious skin cancer.

Male children's genital development issues can be caused by genetic mutations or environmental factors affecting hormone action.

Mouse genital development depends on male or female hormones for specific features.

5α-reductase-2 deficiency causes ambiguous genitalia at birth and affects male sexual development, but individuals often develop male characteristics at puberty.

Androgen and Wnt signals are crucial for developing and functioning male genitalia and erectile tissues.

Insufficient androgen action in male fetuses can cause genital development issues due to genetic mutations or environmental chemicals.

Gene mutations can cause problems in male genital development.

Finasteride causes reproductive malformations in male rats.