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The document concludes that antiandrogenic drugs like cyproterone acetate and spironolactone are effective but not permanent treatments for skin-related androgenization in women.

Lowering testosterone speeds up wound healing in male mice.

Cyproterone acetate treatment is safe and causes mild feminization, which increases with added estrogen.

Certain pesticides and fungicides can interfere with male hormone functions, potentially causing reproductive issues in male rats.

Finasteride affects male rat genitalia development, causing abnormalities during specific pregnancy days.

Blocking androgen receptors early in life increases estrogen levels and reduces sexual motivation in male rats.

Men might have a version of the female disease, polycystic ovarian syndrome, shown by changes in hormone levels and early baldness, but more research is needed to fully understand it.

Antiandrogens affect androgen-dependent body functions and are used for various medical conditions, with some risks like fetus feminization, but new forms like 17α-propylmesterolone show promise for acne without systemic effects.

Men generally face more severe COVID-19 outcomes than women, partly due to hormonal differences.

Fadrozole and Finasteride change frog sex ratios and cause intersex animals with altered gene expressions.

Testosterone can cause acne and male-pattern baldness, affects hair growth in men and women, and makes male skin more sensitive.

Testosterone and some of its forms can strongly stimulate oil gland growth in skin.

Anabolic steroids and inflammation can disrupt androgen metabolism, potentially causing health issues.

Men's sensitivity to male hormones might affect how severe COVID-19 gets for them.

Testosterone changes how certain channels work in male rat reproductive tracts, affecting fertility-related fluid balance.

Cyproterone acetate is a safe treatment that causes mild feminizing effects and is more effective with added estrogens.

Male tissue has more cell death than female tissue after ischemia, and some neurosteroids only protect female cells.

Long-term high testosterone levels can improve bone density and reduce body fat but may increase the risk of prostate cancer and high blood pressure.

Androgen is important in controlling stem cell differentiation, reducing fat development, and increasing lean mass.

Advertisements shape and reinforce idealized masculinity and gender norms.

MENT could be a better option than testosterone for male hormone therapy and birth control because it works well at lower doses and has fewer side effects on the prostate.

Hormone treatment for transsexual individuals is effective but carries risks like thromboembolic events and mood changes, with most side effects being minor and reversible.

Blocking testosterone speeds up wound healing in males.

Finasteride significantly reduced sperm production and caused testicular atrophy in hamsters.

Androgen receptor inactivation delays skin cancer onset and reduces incidence in mice, with gender differences in susceptibility.

Finasteride may cause breast enlargement, low testosterone a risk factor.

Finasteride affects prostate development differently in male and female Mongolian gerbils.

Hormone therapy for trans individuals is effective and generally safe in the short term.