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Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Meis1 is crucial for skin health and tumor development.

848 genes related to fat and metabolism are less active in people with Central Centrifugal Cicatricial Alopecia.

Caloric restriction improves skin and fur structure but can cause muscle loss and movement issues.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.

Different keratins have unique expression patterns in mouse skin cells.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

Blocking the mineralocorticoid receptor can help treat skin thinning caused by steroids.

mTORC2 is crucial for healthy skin barrier by regulating lipids and filaggrin.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

MC-DNA vector-based gene therapy can temporarily treat CBS deficiency in mice.

Blocking mTORC1 activity could increase hair pigmentation and potentially reverse greying.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

The Hairless gene is crucial for healthy skin and hair growth.

Alopecia can be caused by multicentric reticulohistiocytosis.

Retinoids or their analogs could treat skin pigmentation disorders like melasma and vitiligo.

Blocking the JAK/STAT pathway may help reduce skin sensitivity in Xeroderma pigmentosum.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Mitochondrial Complex I reduces inflammation and increases bone breakdown by affecting certain immune cells.

Melanocyte stem cells are crucial for skin pigmentation and have potential in disease modeling and regenerative medicine.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

C-reactive protein helps monocytes stick to blood vessel cells by causing oxidative stress.

BRG1 is essential for skin cells to move and heal wounds properly.

A new ARID1B gene variation causes Coffin-Siris syndrome 1 and early high myopia in a Chinese family.

TCDD exposure causes sebaceous glands to differentiate and produce more lipids before shrinking.