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Nestin marks cells that can become a specific type of skin cell in hair follicles of both developing and adult mice.

MOF controls skin development by regulating genes for mitochondria and cilia.

Nestin-expressing cells turn into a specific type of skin cell in hair follicles during development and in adults.

Tmem50b and 2610305D13Rik genes play key roles in early mouse embryo development.

SOX11 and SOX4 help skin cells act like embryonic cells to heal wounds in mice.

Thyroid hormone and epidermal growth factor affect hair angle, tooth eruption, and ear development in rats.

The study maps how genes are regulated during mouse hair growth.

Reptilian scales, feathers, and hairs evolved from changes in skin cell interactions.

Par3–mInsc and Gαi3 work together to ensure proper cell division orientation in skin development.

Genetically modified cells can regenerate skin and hair in rats.

Fibronectin and other basement membrane components increase during hair growth and decrease during rest.

mEphA1 receptor tyrosine kinase is important for skin and hair development and may play a role in certain diseases.

Keratin expression reflects cell organization and differentiation, not causes it.

Tissue stiffness affects hair follicle regeneration, and Twist1 is a key regulator.

Nestin-expressing progenitor cells become outer root sheath keratinocytes.

Stimulating OR10J1 can promote hair growth and may help treat hair loss.

Root hair growth mechanics depend on turgor pressure and cell wall properties.

Hair growth may involve a pulling force from the outer root sheath.

Combining biochemical, immune, and mechanical signals can improve skin regeneration.

ERK activation spreads between cells, influencing cell division and wound healing.

Hoxc genes control hair growth through Wnt signaling.

Endoglin is important for proper hair growth cycles and stem cell activation in hair follicles.

BMP2 needs periosteal tissue to help regenerate mouse middle finger bones within a specific time.

Dendritic cells help regulate skin development and hair growth in mice.

Activin A and follistatin control when ear hair cells form in mice.

The research concluded that hyaluronic acid affects the formation and growth of hair follicle-like structures in a lab setting.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

The research found how GPCR Class A Rhodopsin receptors are related and suggested possible substances they interact with.