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Melanoma risk tools need improvement, a gene mutation causes a hair disorder that might be treated by managing cell stress, a potential therapy for a skin-ear disorder involves blocking cell channels, skin wrinkling may indicate lung aging regardless of smoking, and oxidative stress might contribute to common baldness.

α-Mangostin can naturally lighten skin by reducing melanin.

A rare pigmented skin tumor called melanotrichoblastoma was diagnosed in a 51-year-old woman.

KIT's role in skin cells is not entirely independent, as other cells can influence its function.

Sulfated hyaluronan in collagen helps hair follicle cells grow and develop better for skin grafts.

The risk of skin tumors becoming malignant depends on the specific skin cell type affected.

Tumor cell adhesion is linked to higher risk of SLN metastasis and melanoma recurrence, and a model including these factors predicts these outcomes better than one with just clinical data.

Melanocytes are destroyed by factors needed for melanin production, causing vitiligo and gray hair.

Melanin and melanocytes contribute to the brown pigmentation in stasis dermatitis.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Hair graying is caused by the loss of pigment cells due to poor maintenance of stem cells in the hair follicle.

K6 gene expression can be controlled and manipulated in mice for studying skin disorders.

Melanogenesis inhibitors like kojic acid and niacinamide can reduce inflammation and pigment production in skin cells.

A specific DNA region controls skin cell gene expression by working with certain proteins.

The PML protein helps prevent skin cancer in mice.

Synthetic melanin applied to skin speeds up wound healing.

Melanocyte transplantation can safely restore skin color, especially in stable vitiligo, but must be chosen carefully based on the disease phase.

Certain VDR gene changes can affect melanoma risk.

Blocking casein kinase 1 in skin cells can help melanocyte precursors move better, potentially helping with conditions like vitiligo or gray hair.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

The model can effectively test gene functions and drug responses in human skin.

Bcl-2 affects hair growth and pigmentation by controlling cell death.

Moles may stop growing because of cell cooperation, not just because of aging cells.

Removing Dicer from pigment cells in newborn mice causes early hair graying and changes in cell migration molecules.

Brown skin in guinea pigs has more ascorbic acid and related enzymes, black skin uses ascorbic acid well, and white skin has the most protein.

A specific gene segment can make mouse skin cells glow, helping study hair growth and gene effects.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Graying hair is caused by changes in gene expression affecting cell functions.

A new genetic model may improve treatment and diagnosis for certain inherited skin diseases.