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Merkel cells develop independently of nerves and are linked to specific hair follicles in mice.

Stress hormone CRF causes hair loss and inhibits hair growth in human cells.

Hair follicle stem cells can turn into various cell types and help repair nerves.

Early and late matrix progenitors in hair follicles create different cell layers, with early ones forming the companion layer and later ones forming the inner root sheath and hair shaft.

Hair follicles can be a good source of stem cells like those from bone marrow.

Human hair follicle cells can help regenerate hair and reduce inflammation, aiding in hair loss treatment.

Merkel cells are more densely found in hairless skin areas like palms and soles.

Basal layer skin cells help form the epidermis and hair follicles.

Certain immune cells are more common on the top of the head and might help predict or treat common hair loss.

Stem and progenitor cells in the eye have different division rates and locations, affecting how they respond to injury.

Stress hormone CRF causes hair loss and stops hair cell growth.

The mineralocorticoid receptor temporarily affects mouse skin development, but the glucocorticoid receptor has a more lasting impact.

Skin cells control immune cell placement, helping the skin respond better to challenges.

Tumor cells in calcifying epithelioma of Malherbe resemble hair follicle cells.

Hair-follicle stem cells can become neurons.

Endocannabinoids help control mast cell activity in human skin.

Merkel cells and Langerhans cells in hair follicles are closely connected and may interact.

Telocytes help organize male reproductive tissues and their changes can lead to diseases.

Hair matrix cysts are rare skin nodules with unique features, often needing surgical removal.

Small molecule DMF improves psoriasis and multiple sclerosis, adult skin cells can be made to grow new hair, certain skin cells initiate hair growth, IL-17C controls gut health and can cause skin inflammation, and skin cells produce IL-17 that can lead to psoriasis.

Cortisol and corticosterone thin mouse skin and regress hair follicles, while testosterone promotes skin and hair development.

Newly divided skin cells quickly move to join skin structures due to tissue tension and specific signals.

Brindled mice show abnormal catecholamine neuron development due to copper deficiency.

Hair and nail cells share similar proteins, indicating a common differentiation pathway.

Different types of skin cells create unique support structures that can affect skin cell growth and could help in skin repair.

Newly born mesenchymal cells quickly spread out in response to tissue tension during early development.

Hair follicles can be used to easily create neurons and glial cells for potential nerve repair.

Activating Sonic Hedgehog signaling in cancer stroma may help treat basal cell carcinoma.