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Stem cell behavior varies with stimuli, and lineage changes can happen without affecting stem cell division.

Epidermolytic ichthyosis can be inherited in a semidominant way with mild symptoms in carriers.

Overexpression of E2F1 can lead to skin tumors and disrupt hair growth.

Mutant cells in hair follicles are influenced by their location and interactions with surrounding cells.

Atoh1 expression can create new Merkel cells in the skin.

A new method can detect mutations in mice by observing changes in hair follicle cells.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.

Tabby mutations in mice affect hair follicle development and help study genetic mapping and certain medical conditions.

Mettl3 is essential for normal tissue development and self-renewal by regulating gene expression.

Meis1 is crucial for skin health and tumor development.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

Using a combination of specific cell cycle regulators is better for safely keeping hair root cells alive indefinitely compared to cancer-related methods.

A mutation in the KRT86 gene causes hair fragility in a Turkish family.

Keratins K6 and K16 are expressed more freely in regenerating mouse skin than K1 and K10.

Msx2-deficient mice experience irregular hair growth and loss due to disrupted hair cycle phases.

Inactivating both p53 and Rb genes in mice speeds up aggressive skin cancer development.

Hair follicles protect melanocytes from sun damage, helping them replenish skin.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

E6/E7 oncogenes in hair follicles cause continuous hair growth by skipping the resting phase.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Fibroblasts from healthy donors can prevent changes seen in recessive epidermolysis bullosa simplex.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.

A common mutation in the hHb6 gene is linked to monilethrix, but other factors may also play a role.

EBV infection increases a specific keratin variant in carcinoma cells, possibly affecting cell structure and cancer progression.