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The TRPV3 channel structure changes linked to severe itch and hyperkeratosis were identified using cryo-EM.

Basement membrane changes are crucial for hair follicle development.

Defective protein folding due to a mutation is key in ANE syndrome.

Women with alopecia areata have poor blood flow, possibly due to nerve inflammation.

A new hydrogel made from human cells improves wound healing by working with immune cells to promote repair.

The FRZB gene slows hair growth in rabbits.

Mechanical stress causes ligament thickening through WISP-1 and Hedgehog signaling.

Hair proteins have weak spots in their α-helical segments.

Particles around 100 nm can penetrate and stay in hair follicles without passing through healthy skin, making them safe for use in topical products and useful for targeted drug delivery.

A 46-year-old man was diagnosed with frontal fibrosing alopecia, a condition usually seen in postmenopausal women.

Cashmere fiber diameter in Tibetan goats is influenced by their stress, oxygen, and metabolic adaptations.

ANE syndrome is caused by a mutation in the RBM28 protein that disrupts ribosome assembly.

Meibomian glands are like hair follicles without a hair shaft.

ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

Lower PPARγ levels and specific gene variations are linked to more severe Frontal Fibrosing Alopecia.

A new protein, mK6irs, is found in specific hair layers and may help understand hair growth and diseases.

Profilin1 speeds up wound healing.

Hair growth drugs and epidermal growth factor do not change the calcium binding site gradient in hair follicles.

The protein structures in the hair and tendon were preserved, but their molecular arrangements changed.

Telomerase-positive cells are mainly in the bulb matrix and outer root sheath of hair follicles.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Ift20 is essential for hair follicle function and skin cell movement.

Connective tissue nevi have distinct features, and reflectance confocal microscopy is useful for early diagnosis.

Scientists found out how a specific protein in human hair cuticles behaves and is structured.

PINCH-1 is crucial for skin cell adhesion and movement, working with EPLIN and ILK.

Targeting specific cell interactions may help treat skin fibrosis.

Myofibroblasts in rat wound healing may come from blood vessel pericytes and perifollicular dermal sheath cells.