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The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

Folliculin deficiency causes problems with cell division and positioning due to disrupted RhoA signaling and interaction with p0071.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

The article's correction clarifies scientific and methodological concerns about miR-200c-3p's role in alopecia areata.

FP-1 is a key protein in rat hair growth, active only during the growth phase.

The early genes of a specific virus can cause abnormal skin cell growth and hair follicle changes.

Lack of TG2 increases fat storage and lowers cell cleanup in skin oil cells.

NFIB and STAT5 work together to control specific genetic programs in cells.

FOXC1 boosts SFRP1 in hair loss, suggesting new treatments.

A protein called HIF-1a helps control hair growth genes and could be targeted to treat hair loss.

FGF5 gene mutations cause unusually long eyelashes by affecting hair growth regulation.

ME1 and PPAR signaling may influence hair loss in androgenetic alopecia.

Eyelid cells share signaling components but differ in pathway activity.

Children with alopecia areata, especially with Down syndrome or thyroid issues, should be screened for thyroid disease.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

The STIM1 R304W mutation in mice leads to bone changes and teeth hair growth.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

Blocking miR-27a increases sheep hair follicle stem cell growth and decreases cell death, which could help improve wool quality and treat hair loss.

An 8-year-old Saudi boy was diagnosed with a rare genetic disorder causing hair loss, skin issues, and light sensitivity.

AMT may cause hair loss and changing dWAT activity could help treat it.

Noggin promotes skin tumors by activating certain cell signaling pathways.

The retinoid receptor antagonist effectively disrupts vitamin A-related development in embryos.

Eating vitamin A affects hair growth and health by changing cell signals in mice.

Bmpr2 and Acvr2a receptors are crucial for hair retention and color.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.

Epimorphin helps shape and develop epithelial cells, like those in hair follicles.