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A specific hair protein variant increases the spread of breast cancer and is linked to worse survival rates.

c-Maf positive macrophages help form hypertrophic scars by affecting fibroblasts and collagen production.

Researchers found a new gene variant linked to a rare bone disease, which doesn't always cause symptoms in carriers.

lncRNA MTC affects protein levels in goat skin cells, impacting hair growth.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.

MTS24 marks a new type of skin cell that helps hair growth and repair.

Tofacitinib helps mice grow more hair by increasing noggin and BMP4 levels, possibly better than minoxidil.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Truncated LTBP-1 disrupts TGF-beta signaling, affecting hair growth.

MAP-2 is crucial for the structure of hair follicles and nails.

Matriptase-2 helps control iron levels by suppressing hepcidin, and its deficiency can cause iron-deficiency anemia.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

Patients with severe alopecia areata have higher levels of MIF, which decrease after successful treatment.

Acne patients have higher mTOR gene expression.

Some skin growths with mucin can form hair follicles and resemble skin cancer, but a special stain can help tell them apart.

Overexpressing the human H-ferritin gene in mice causes mild growth delay and temporary hair loss.

Skin tumor cells in patients with tuberous sclerosis have higher levels of a protein called cathepsin B.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

MC-1R in skin cells may influence inflammation and collagen production.

The Itpr3 gene causes a specific hair pattern in mice.

Activated LEF/TCF complexes are crucial for hair development and cycling.

Mettl3 is essential for normal tissue development and self-renewal by regulating gene expression.

LTBP1 is a key regulator in diseases and a potential target for new treatments.

miR-3606-3p reduces skin fibrosis by blocking key signaling pathways.

Healthy mitochondria in skin cells are essential for proper hair growth and skin cell interaction in mice.

Transferrin receptor expression increases iron in mouse skin cells without causing damage.

TMPRSS6 is crucial for controlling hepcidin and normal iron absorption.

Truncated LTBP-1 disrupts TGF-β signaling, affecting hair growth.