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Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

New mutations in the SLC39A4 gene found in twins help understand the genetic cause of acrodermatitis enteropathica.

Topical tofacitinib is effective in promoting hair growth for non-scarring alopecia.

A mutation in the KRT25 gene causes a rare hair disorder with thin, woolly hair.

New technologies show potential for better understanding and treating skin conditions with abnormal mucin, but more research is needed for clinical use.

Tofacitinib helped regrow hair in most patients with severe hair loss.

A KRT32 gene variant causes loose anagen hair syndrome.

High MUC-18/MCAM levels in blood indicate a worse outlook for melanoma patients.

IFAP syndrome is a rare genetic disorder causing skin, hair, and eye issues, with limited treatment options.

A mutation in the Sgkl gene causes defective hair growth in mice.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Different hair fiber development might explain why hair loss severity varies in patients with a specific genetic mutation, and treatments that thicken hair could help.

New genetic variants linked to inherited ichthyoses were identified, offering insights for potential gene therapy.

Radiation treatment causes skin fibrosis by increasing certain fibroblast subpopulations, but using a c-Jun inhibitor or fat grafting can reduce this effect.

Monitoring TGF-β and linc-PINT expression may help identify and treat high-risk heart arrhythmia patients.

The first Japanese case of a genetic hair disorder caused by specific mutations in the LIPH gene was identified.

A new mutation linked to partial Androgen Insensitivity Syndrome and prostate cancer was found in a patient unhappy with their female gender assignment.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Tofacitinib improved arthritis and partially improved hair loss in a lupus patient without side effects.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

Mutations in the AP1B1 gene cause a new syndrome with skin, hearing, and developmental issues.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

Two microRNAs affect hair follicle development in sheep by targeting specific genes.

Kidney cancer cells without folliculin are more sensitive to radiation due to increased self-eating cell death.

Some Greek melanoma patients have gene mutations linked to increased cancer risk, a new color feature helps diagnose melanoma, the incidence of a skin condition in the Netherlands is rare, and a gene possibly affects male-pattern baldness.

A mutation in the human hairless gene causes alopecia universalis.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

Mutations in the POC1A gene can cause a unique form of extreme insulin resistance and short stature.