Search

everythinglearnresearchcommunity

for

Search:↓

The study developed a 3D model that closely imitates remaining ovarian cancer after treatment and identified a potential drug targeting resistant cancer cells.

A new mutation in the hairless gene causes hair loss and skin wrinkling in mice.

Injury boosts normal skin cell growth, reducing cancer cell advantage.

Nε-(carboxymethyl) lysine delays hair growth by blocking a key protein.

The PML protein helps prevent skin cancer in mice.

CRISPR/Cas9 editing in spinach affects root hair growth by altering specific genes.

A new method quickly creates controllable cell clusters for tissue engineering and drug testing.

Activating certain cells in hair follicles can prevent hair loss caused by cancer treatments.

The tool iCOUNT helps understand how stem cells divide and affect tissue development and repair.

The Kras mutation changes normal cell signals, leading to disrupted tissue structure and potential cancer.

Cathepsin L deficiency causes hair and skin issues in mice.

Manipulating cell sorting can improve hair follicle regeneration in the lab.

The treatments stopped hair regrowth in mice.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

Reduced matriptase activity causes skin and hair issues in both humans and mice.

Tabby mutations in mice affect hair follicle development and help study genetic mapping and certain medical conditions.

Cells lacking the Bax protein can outcompete others, leading to better tissue repair and hair growth.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

The study concludes that a genetic mutation in TFM mice leads to reduced androgen receptor activity, affecting the body's response to male hormones.

Myc activation reduces skin stem cells by affecting cell adhesion.

Matriptase is crucial for skin, hair, and immune cell health, and its imbalance can lead to cancer.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

Defective mitochondrial DNA replication causes aging symptoms and increased oxidative damage in mice.

Blocking hair follicle development stops key gene signals needed for hair growth in mice.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Identical twins had different symptoms because one had more cells with an extra chromosome fragment in different tissues.

Mutating the gmds gene in zebrafish increases hair cell numbers and regeneration.

Matriptase is crucial for skin barrier, hair growth, and may contribute to skin cancer.