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Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

Modified β-catenin can cause different effects in mouse skin cells, leading to cysts or tumors depending on the cell type.

Dopaminergic neurons in the gut have diverse subtypes with different neurotransmitter contents.

FoxN1 overexpression in young mice harms immune cell and skin development.

HLD10 can include increased body hair and Mongolian spots.

Defective protein folding due to a mutation is key in ANE syndrome.

Overexpression of ΔNLef1 in mouse skin leads to hair loss, cysts, and skin tumors.

Antizyme slows skin tumor growth by reducing cell growth in mice.

Injecting certain cells into mice caused hair loss, which was preventable with a specific inhibitor.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Removing MCPIP1 from myeloid cells in mice leads to hair loss and prevents skin tumors but causes pigmented spots.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Removing the ATR gene in adult mice causes rapid aging and stem cell loss.

A new MBTPS2 gene variant disrupts fat metabolism and collagen production, causing Osteogenesis imperfecta.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

Secreted inhibitors of Wnt and IGF signaling control hair and tooth development, creating species-specific patterns.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

New mouse models of Pemphigus show severe symptoms and need better treatments.

Minoxidil treatment improves heart defects in a DiGeorge syndrome model.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

A deletion in the CDH3 gene causes a rare disorder with short hair and vision loss.

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

Mutations in the Sgk3 gene cause fuzzy hair in mice.

Bezafibrate treatment improved skin and spleen health in aging mice but didn't extend lifespan.

ErbB2 signaling is crucial for skin cell growth and cancer development in mice.